Prostate cancer cells differ in testosterone accumulation, dihydrotestosterone conversion, and androgen receptor signaling response to steroid 5α-reductase inhibitors-Reference-Cited by-同舟云学术

Prostate cancer cells differ in testosterone accumulation, dihydrotestosterone conversion, and androgen receptor signaling response to steroid 5α-reductase inhibitors

Published:2013-06-27 Issue:13 Volume:73 Page:1470-1482
ISSN:0270-4137
Container-title:The Prostate
language:en
Short-container-title:Prostate

Author:

Wu Yue¹²,Godoy Alejandro²³,Azzouni Faris²,Wilton John H.⁴,Ip Clement¹,Mohler James L.²⁵

Affiliation:

1. Department of Cancer Prevention and Control; Roswell Park Cancer Institute; Buffalo New York

2. Department of Urology; Roswell Park Cancer Institute; Buffalo New York

3. Department of Physiology; Pontificia Universidad Católica de Chile; Santiago de Chile

4. Department of Medicine; Roswell Park Cancer Institute; Buffalo New York

5. Department of Urology; University at Buffalo School of Medicine and Biotechnology; Buffalo New York

Funder

National Cancer Institute

National Cancer Institute, Cancer Center

Publisher

Wiley

Subject

Urology,Oncology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/pros.22694/fullpdf

Reference38 articles.

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2. Steroid 5 alpha-reductase: Two genes/two enzymes;Russell;Annu Rev Biochem,1994

3. Modulation of androgen receptor activation function 2 by testosterone and dihydrotestosterone;Askew;J Biol Chem,2007

4. Binding properties of androgen receptors. Evidence for identical receptors in rat testis, epididymis, and prostate;Wilson;J Biol Chem,1976

5. Castration-recurrent prostate cancer is not androgen-independent;Mohler;Adv Exp Med Biol,2008

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