Priming of LTP in amygdala and hippocampus by prior paired pulse facilitation paradigm in mice lacking brain serotonin

Author:

Gebhardt Christine1,Mosienko Valentina2,Alenina Natalia23,Albrecht Doris1ORCID

Affiliation:

1. Institute of Neurophysiology, Charité ‐ Universitätsmedizin Berlin Berlin Germany

2. Max‐Delbrück Center for Molecular Medicine in the Helmholtz Association Berlin Germany

3. Institute of Translational Biomedicine, St. Petersburg State University St. Petersburg Russia

Publisher

Wiley

Subject

Cognitive Neuroscience

Reference73 articles.

1. Metaplasticity: The plasticity of synaptic plasticity;Abraham W. C.;Trends in Neurosciences,1996

2. Growth retardation and altered autonomic control in mice lacking brain serotonin;Alenina N.;Proceedings of the National Academy of Sciences of the United States of America,2009

3. Mice genetically depleted of brain serotonin do not display a depression‐like behavioral phenotype;Angoa‐Perez M.;ACS Chemical Neuroscience,2014

4. Araragi N.(2013)Electrophysiological investigation of two animal models for emotional disorders: Serotonin transporter knockout mice and tryptophan hydroxylase 2 knockout mice. Medizinische Fakultät Klinik und Poliklinik für Psychiatrie Psychosomatik und Psychotherapie. Thesis Universität Würzburg 1–164.

5. The catecholaminergic innervation of the rat amygdala;Asan E.;Advances in Anatomy, Embryology, and Cell Biology,1998

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