Experimental myasthenia: Lack of correlation between the autoantibody titer and the reduction of acetylcholine-controlled ionic channels measured at functioning endplates
Author:
Publisher
Wiley
Subject
Physiology (medical),Cellular and Molecular Neuroscience,Clinical Neurology,Physiology
Reference18 articles.
1. Properties of end-plate channels in rats immunized against acetylcholine receptors
2. Myasthenia induced by monoclonal anti-acetylcholine receptor antibodies: Clinical and electrophysiological aspects
3. The Biology of Myasthenia Gravis
4. Functional Activities of Autoantibodies to Acetylcholine Receptors and the Clinical Severity of Myasthenia Gravis
5. Determination of dose-response curves by quantitative ionophoresis at the frog neuromuscular junction.
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1. IL-4 receptor as a bridge between the immune system and muscle in experimental myasthenia gravis I;Clinical Immunology;2009-08
2. Short-circuiting autoimmune disease by target-tissue-derived nitric oxide;Clinical Immunology;2004-10
3. Myocyte production of nitric oxide in response to AChR-reactive antibodies in two inbred rat strains may influence disease outcome in experimental myasthenia gravis;Clinical Immunology;2003-02
4. Muscle Responds to an Antibody Reactive with the Acetylcholine Receptor by Up-Regulating Monocyte Chemoattractant Protein 1: A Chemokine with the Potential to Influence the Severity and Course of Experimental Myasthenia Gravis;The Journal of Immunology;2002-08-01
5. Myocytes respond in vivo to an antibody reactive with the acetylcholine receptor by upregulating interleukin-15: an interferon-gamma activator with the potential to influence the severity and course of experimental myasthenia gravis;Journal of Neuroimmunology;2001-10
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