Cerebral amyloid‐β load is associated with neurodegeneration and gliosis: Mediation by p‐tau and interactions with risk factors early in the Alzheimer's continuum

Author:

Salvadó Gemma12,Milà‐Alomà Marta1234,Shekari Mahnaz123,Minguillon Carolina124,Fauria Karine14,Niñerola‐Baizán Aida56,Perissinotti Andrés56,Kollmorgen Gwendlyn7,Buckley Christopher8,Farrar Gill8,Zetterberg Henrik9101112,Blennow Kaj910,Suárez‐Calvet Marc12413,Molinuevo José Luis1,Gispert Juan Domingo1236,

Affiliation:

1. Barcelonaβeta Brain Research Center (BBRC) Pasqual Maragall Foundation Barcelona Spain

2. IMIM (Hospital del Mar Medical Research Institute) Barcelona Spain

3. Universitat Pompeu Fabra Barcelona Spain

4. Centro de Investigación Biomédica en Red de Fragilidad y Envejecimiento Saludable (CIBERFES) Madrid Spain

5. Nuclear Medicine Department Hospital Clínic Barcelona Barcelona Spain

6. Centro de Investigación Biomédica en Red Bioingeniería, Biomateriales y Nanomedicina, (CIBER‐BBN) Barcelona Spain

7. Roche Diagnostics GmbH Penzberg Germany

8. GE Healthcare Life Sciences Amersham UK

9. Department of Psychiatry and Neurochemistry Institute of Neuroscience and Physiology University of Gothenburg Mölndal Sweden

10. Clinical Neurochemistry Laboratory Sahlgrenska University Hospital Mölndal Sweden

11. Department of Neurodegenerative Disease UCL Institute of Neurology Queen Square London UK

12. UK Dementia Research Institute at UCL London UK

13. Servei de Neurologia Hospital del Mar Barcelona Spain

Publisher

Wiley

Subject

Psychiatry and Mental health,Cellular and Molecular Neuroscience,Geriatrics and Gerontology,Neurology (clinical),Developmental Neuroscience,Health Policy,Epidemiology

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