Killer-Cell Immunoglobulin-Like Receptors (KIR) and HLA-Class I Heavy Chains in Ankylosing Spondylitis
Author:
Affiliation:
1. Rheumatology Unit; Department of Medical Sciences; University of Cagliari; Cagliari 090142 Italy
2. Haematology Unit; Department of Medical Sciences; University of Cagliari; Cagliari 090142 Italy
Publisher
Wiley
Subject
Drug Discovery
Reference24 articles.
1. Th17 cells expressing KIR3DL2+ and responsive to HLA-B27 homodimers are increased in ankylosing spondylitis;Bowness;J Immunol,2011
2. Association scan of 14,500 non-synonymous SNPs in four diseases identifies autoimmunity variants;Burton;Nat Genet,2007
3. Analysis of the HLA-B27 extended haplotypes in Sardinia discloses additional predisposing factors for ankylosing spondylitis;Cascino;Arthritis Rheum,2007
4. Th17 and interleukin 23 in the pathogenesis of psoriatic arthritis and spondyloarthritis;Cauli;J Rheumatol Suppl,2012b
5. Increased level of HLA-B27 expression in ankylosing spondylitis patients compared with healthy HLA-B27 subjects: a possible further susceptibility factor for the development of disease;Cauli;Rheumatology,2002a
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