The involvement of thrombin in the pathogenesis of glioblastoma

Author:

Krenzlin Harald12,Lorenz Viola3,Alessandri Beat2

Affiliation:

1. Harvey Cushing Neuro-oncology Laboratories, Department of Neurosurgery; Brigham and Women's Hospital, Harvard Medical School; Boston MA 02115 USA

2. Institute for Neurosurgical Pathophysiology; University Medicine Mainz; Langenbeckstr.1 Mainz 55101 Germany

3. Division of Newborn Medicine, Boston Children's Hospital; 300 Longwood Avenue Boston MA 02115 USA

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Reference53 articles.

1. How much is enough? The question of extent of resection in glioblastoma multiforme;Abd-El-Barr;World Neurosurg,2014

2. PAR1 inhibition suppresses the self-renewal and growth of A2B5-defined glioma progenitor cells and their derived gliomas in vivo;Auvergne;Oncogene,2016

3. Protease nexin-1. Localization in the human brain suggests a protective role against extravasated serine proteases;Choi;Am J Pathol,1990

4. Molecular mechanisms of thrombin signaling;Coughlin;Semin Hematol,1994a

5. Protease-activated receptors start a family;Coughlin;Proc Natl Acad Sci U S A,1994b

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