Melatonin reverses the oxidative stress and mitochondrial dysfunction caused by LETM1 silencing
Author:
Affiliation:
1. Department of Molecular Biology and Genetics, Faculty of Science and Arts Namık Kemal University 59030 Tekirdağ Turkey
Publisher
Wiley
Subject
Cell Biology,General Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/cbin.11274
Reference36 articles.
1. Melatonin, Mitochondrial Homeostasis and Mitochondrial-Related Diseases
2. Melatonin-mitochondria Interplay in Health and Disease
3. Strain-dependent effects of long-term treatment with melatonin on kainic acid-induced status epilepticus, oxidative stress and the expression of heat shock proteins
4. LETM1: Essential for Mitochondrial Biology and Cation Homeostasis?
5. LETM1-Mediated K+ and Na+ Homeostasis Regulates Mitochondrial Ca2+ Efflux
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1. LETM1: A Single Entity With Diverse Impact on Mitochondrial Metabolism and Cellular Signaling;Frontiers in Physiology;2021-03-18
2. The leucine zipper EF‐hand containing transmembrane protein‐1 EF‐hand is a tripartite calcium, temperature, and pH sensor;Protein Science;2021-03-02
3. Glucocorticoid receptors involved in melatonin inhibiting cell apoptosis and NLRP3 inflammasome activation caused by bacterial toxin pyocyanin in colon;Free Radical Biology and Medicine;2021-01
4. Preventive treatments to slow substantia nigra damage and Parkinson’s disease progression: A critical perspective review;Pharmacological Research;2020-11
5. Melatonin inhibits the apoptosis of rooster Leydig cells by suppressing oxidative stress via AKT-Nrf2 pathway activation;Free Radical Biology and Medicine;2020-11
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