Periosteal Mesenchymal Progenitor Dysfunction and Extraskeletally‐Derived Fibrosis Contribute to Atrophic Fracture Nonunion-Reference-Cited by-同舟云学术

Periosteal Mesenchymal Progenitor Dysfunction and Extraskeletally‐Derived Fibrosis Contribute to Atrophic Fracture Nonunion

Published:2019-01-02 Issue:3 Volume:34 Page:520-532
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Wang Luqiang¹²,Tower Robert J¹,Chandra Abhishek¹,Yao Lutian¹³,Tong Wei¹⁴,Xiong Zekang⁴,Tang Kai⁴,Zhang Yejia¹⁵⁶,Liu X Sherry¹,Boerckel Joel D¹⁷,Guo Xiaodong⁴,Ahn Jaimo¹,Qin Ling¹

Affiliation:

1. Department of Orthopaedic SurgeryPerelman School of MedicineUniversity of PennsylvaniaPhiladelphiaPAUSA

2. Department of OrthopaedicsShandong University Qilu HospitalShandong UniversityJinanChina

3. Department of Orthopaedics/Sports Medicine and Joint SurgeryThe First Hospital of China Medical UniversityShenyangChina

4. Department of OrthopaedicsUnion HospitalTongji Medical CollegeHuazhong University of Science and TechnologyWuhanChina

5. Department of Physical Medicine and RehabilitationPerelman School of MedicineUniversity of PennsylvaniaPhiladelphiaPAUSA

6. Translational Musculoskeletal Research Center (TMRC)Corporal Michael J. Crescenz Veterans Affairs Medical CenterPhiladelphiaPAUSA

7. Department of BioengineeringUniversity of PennsylvaniaPhiladelphiaPAUSA

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbmr.3626

Reference37 articles.

1. Fracture healing: mechanisms and interventions

2. Prevalence of long-bone non-unions

3. Delayed union and nonunions: Epidemiology, clinical issues, and financial aspects

4. Mice expressing GFP and CreER in osteochondro progenitor cells in the periosteum

5. Use of an alpha-smooth muscle actin GFP reporter to identify an osteoprogenitor population

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