Epithelial barrier disruption allows nondisease-causing bacteria to initiate and sustain IBD in the IL-10 gene-deficient mouse-Reference-Cited by-同舟云学术

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Epithelial barrier disruption allows nondisease-causing bacteria to initiate and sustain IBD in the IL-10 gene-deficient mouse

Published:2007-08 Issue:8 Volume:13 Page:947-954
ISSN:1078-0998
Container-title:Inflammatory Bowel Diseases
language:en
Short-container-title:Inflammatory Bowel Diseases

Author:

Sydora Beate C.,MacFarlane Sarah M.,Walker John W.,Dmytrash Andrea L.,Churchill Thomas A.,Doyle Jason,Fedorak Richard N.

Publisher

Oxford University Press (OUP)

Subject

Gastroenterology,Immunology and Allergy

Reference27 articles.

1. Helicobacter hepaticus Does Not Induce or Potentiate Colitis in Interleukin-10-Deficient Mice

2. Enterococcus faecalis Induces Inflammatory Bowel Disease in Interleukin-10 Knockout Mice

3. Variable phenotypes of enterocolitis in interleukin 10-deficient mice monoassociated with two different commensal bacteria

4. Association with Selected Bacteria Does Not Cause Enterocolitis in IL-10 Gene-Deficient Mice Despite a Systemic Immune Response

5. Side effects of nonsteroidal anti-inflammatory drugs on the small and large intestine in humans

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