Kinesin family member 6 (kif6) is necessary for spine development in zebrafish

Author:

Buchan Jillian G.1,Gray Ryan S.2,Gansner John M.3,Alvarado David M.4,Burgert Lydia5,Gitlin Jonathan D.6,Gurnett Christina A.457,Goldsmith Matthew I.15

Affiliation:

1. Department of Genetics; Washington University School of Medicine; St. Louis Missouri

2. Department of Developmental Biology; Washington University School of Medicine; St. Louis Missouri

3. Dana-Farber Cancer Institute; Boston Massachusetts

4. Department of Orthopaedic Surgery; Washington University School of Medicine; St. Louis Missouri

5. Department of Pediatrics; Washington University School of Medicine; St. Louis Missouri

6. Eugene Bell Center for Regenerative Biology; Marine Biological Laboratory; Woods Hole Massachusetts

7. Department of Neurology; Washington University School of Medicine; St. Louis Missouri

Funder

Children's Discovery Institute

Publisher

Wiley

Subject

Developmental Biology

Reference51 articles.

1. Lack of association between the Trp719Arg polymorphism in kinesin-like protein-6 and coronary artery disease in 19 case-control studies;Assimes;J Am Coll Cardiol,2010

2. The ciliopathy gene cc2d2a controls zebrafish photoreceptor outer segment development through a role in Rab8-dependent vesicle trafficking;Bachmann-Gagescu;Hum Mol Genet,2011

3. Five common gene variants identify elevated genetic risk for coronary heart disease;Bare;Genet Med,2007

4. Efficient mapping and cloning of mutations in zebrafish by low-coverage whole-genome sequencing;Bowen;Genetics,2012

5. Etiologic theories of idiopathic scoliosis: the breaking of bilateral symmetry in relation to left-right asymmetry of internal organs, right thoracic adolescent idiopathic scoliosis (AIS) and vertebrate evolution;Burwell;Stud Health Technol Inform,2006

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