The Role of Intestinal C‐type Regenerating Islet Derived‐3 Lectins for Nonalcoholic Steatohepatitis-Reference-Cited by-同舟云学术

The Role of Intestinal C‐type Regenerating Islet Derived‐3 Lectins for Nonalcoholic Steatohepatitis

Published:2018-02-28 Issue:4 Volume:2 Page:393-406
ISSN:2471-254X
Container-title:Hepatology Communications
language:en
Short-container-title:Hepatology Communications

Author:

Bluemel Sena¹^ORCID,Wang Lirui¹²,Martino Cameron³,Lee Suhan¹,Wang Yanhan¹²,Williams Brandon¹,Horvath Angela⁴⁵,Stadlbauer Vanessa⁴,Zengler Karsten³⁶,Schnabl Bernd¹²

Affiliation:

1. Department of MedicineUniversity of California San DiegoLa Jolla CA

2. Department of MedicineVA San Diego Healthcare SystemSan Diego CA

3. Department of PediatricsDivision of Host‐Microbe Systems and TherapeuticsSan Diego CA

4. Department of Internal Medicine, Division of Gastroenterology and HepatologyMedical University of GrazGraz Austria

5. Center of Biomarker Research in MedicineGraz Austria

6. Center for Microbiome InnovationUniversity of California San DiegoLa Jolla CA

Funder

Swiss National Science Foundation

National Institutes of Health

Biomedical Laboratory Research and Development Service of the VA Office of Research and Development

Publisher

Wiley

Subject

Hepatology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/hep4.1165

Reference50 articles.

1. Cause, Pathogenesis, and Treatment of Nonalcoholic Steatohepatitis

2. Intestinal microbiota contributes to individual susceptibility to alcoholic liver disease

3. The severity of nonalcoholic fatty liver disease is associated with gut dysbiosis and shift in the metabolic function of the gut microbiota

4. Deficiency of intestinal mucin-2 protects mice from diet-induced fatty liver disease and obesity

5. Intestinal microbiota determines development of non-alcoholic fatty liver disease in mice

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