Molecular Dissection of Somatic Skeletal Disease in Neurofibromatosis Type 1-Reference-Cited by-同舟云学术

Molecular Dissection of Somatic Skeletal Disease in Neurofibromatosis Type 1

Published:2022-12-13 Issue:2 Volume:38 Page:288-299
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J of Bone & Mineral Res

Author:

Paria Nandina¹,Khalid Aysha¹,Shen Bo²,Lemoine Ben³,Chan Jinyan³⁴,Kidane Yared H¹,Oxendine Ila¹,Cornelia Reuel¹,Wise Carol A¹⁵⁶⁷,Rios Jonathan J¹⁵⁶⁷^ORCID

Affiliation:

1. Center for Pediatric Bone Biology and Translational Research, Scottish Rite for Children Dallas TX USA

2. Children's Research Institute, UT Southwestern Medical Center Dallas TX USA

3. Baylor Scott & White Research Institute Dallas TX USA

4. Institute of Biomedical Studies, Baylor University Waco TX USA

5. McDermott Center for Human Growth and Development, UT Southwestern Medical Center Dallas TX USA

6. Department of Pediatrics UT Southwestern Medical Center Dallas TX USA

7. Department of Orthopaedic Surgery UT Southwestern Medical Center Dallas TX USA

Funder

National Institutes of Health

U.S. Department of Defense

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbmr.4755

Reference50 articles.

1. Osteogenesis depends on commissioning of a network of stem cell transcription factors that act as repressors of adipogenesis

2. Mapping Distinct Bone Marrow Niche Populations and Their Differentiation Paths

3. Leptin Receptor Promotes Adipogenesis and Reduces Osteogenesis by Regulating Mesenchymal Stromal Cells in Adult Bone Marrow

4. Combined MEK Inhibition and BMP2 Treatment Promotes Osteoblast Differentiation and Bone Healing inNf1Osx−/−Mice

5. FGFR3 in Periosteal Cells Drives Cartilage-to-Bone Transformation in Bone Repair

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