Platelet‐Derived Growth Factor B Is a Key Element in the Pathological Bone Formation of Ankylosing Spondylitis-Reference-Cited by-同舟云学术

Platelet‐Derived Growth Factor B Is a Key Element in the Pathological Bone Formation of Ankylosing Spondylitis

Published:2022-12-09 Issue:2 Volume:38 Page:300-312
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J of Bone & Mineral Res

Author:

Jo Sungsin¹^ORCID,Lee Seung Hoon¹,Park Jinsung¹,Nam Bora¹²,Kim Hyunsung³,Youn Jeehee⁴,Lee Seunghun⁵,Kim Tae‐Jong⁶,Sung Il‐Hoon⁷,Choi Sung Hoon⁷,Park Ye‐Soo⁸^ORCID,Inman Robert D⁹,Kim Tae‐Hwan¹²^ORCID

Affiliation:

1. Hanyang University Institute for Rheumatology Research (HYIRR), Hanyang University Seoul Republic of Korea

2. Department of Rheumatology Hanyang University Hospital for Rheumatic Diseases Seoul Republic of Korea

3. Department of Pathology Hanyang University Hospital Seoul Republic of Korea

4. Department of Anatomy and Cell Biology, College of Medicine Hanyang University Seoul Republic of Korea

5. Department of Radiology Hanyang University Hospital for Rheumatic Disease Seoul Republic of Korea

6. Department of Rheumatology Chonnam National University Medical School and Hospital Gwangju Republic of Korea

7. Department of Orthopaedic Surgery Hanyang University Hospital Seoul Republic of Korea

8. Department of Orthopedic Surgery, Guri Hospital Hanyang University College of Medicine Guri Republic of Korea

9. Shroeder Arthritis Institute, University Health Network, University of Toronto Toronto Canada

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbmr.4751

Reference43 articles.

1. Axial spondyloarthritis

2. Pathogenesis of ankylosing spondylitis — recent advances and future directions

3. Bone Disease in Axial Spondyloarthritis

4. Progress in Spondylarthritis. Mechanisms of new bone formation in spondyloarthritis

5. Tenascin-C-mediated suppression of extracellular matrix adhesion force promotes entheseal new bone formation through activation of Hippo signalling in ankylosing spondylitis

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