Paradoxical Effects of Endoplasmic Reticulum Aminopeptidase 1 Deficiency on <scp>HLA</scp> – <scp>B27</scp> and Its Role as an Epistatic Modifier in Experimental Spondyloarthritis-Reference-Cited by-同舟云学术

Paradoxical Effects of Endoplasmic Reticulum Aminopeptidase 1 Deficiency on HLA – B27 and Its Role as an Epistatic Modifier in Experimental Spondyloarthritis

Published:2022-12-28 Issue:2 Volume:75 Page:220-231
ISSN:2326-5191
Container-title:Arthritis & Rheumatology
language:en
Short-container-title:Arthritis & Rheumatology

Author:

Tran Tri M.¹,Gill Tejpal¹^ORCID,Bennett Joshua¹,Hong Sohee¹,Holt Vance¹,Lindstedt Anders J.¹,Bakshi Sufia¹,Sikora Keith¹,Taurog Joel D.²,Breban Maxime³,Navid Fatemeh¹,Colbert Robert A.¹^ORCID

Affiliation:

1. Pediatric Translational Research Branch, National Institute of Arthritis and Musculoskeletal and Skin Diseases, NIH Bethesda Maryland

2. Division of Rheumatic Diseases, Department of Medicine University of Texas Southwestern Medical Center Dallas

3. Infection and Inflammation Department, UMR1173, INSERM, Université de Versailles Saint‐Quentin‐en‐Yvelines/Université Paris Saclay, and Rheumatology Department, Ambroise Paré Hospital Paris France

Funder

The National Institute of Arthritis and Musculoskeletal and Skin Diseases

Publisher

Wiley

Subject

Immunology,Rheumatology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/art.42327

Reference51 articles.

1. Association scan of 14,500 nonsynonymous SNPs in four diseases identifies autoimmunity variants

2. Genome-wide association analysis identifies new susceptibility loci for Behçet's disease and epistasis between HLA-B*51 and ERAP1

3. A genome-wide association study identifies new psoriasis susceptibility loci and an interaction between HLA-C and ERAP1