Suppression of reactive oxygen species-mediated ERK and JNK activation sensitizes dihydromyricetin-induced mitochondrial apoptosis in human non-small cell lung cancer-Reference-Cited by-同舟云学术

Suppression of reactive oxygen species-mediated ERK and JNK activation sensitizes dihydromyricetin-induced mitochondrial apoptosis in human non-small cell lung cancer

Published:2016-08-19 Issue:4 Volume:32 Page:1426-1438
ISSN:1520-4081
Container-title:Environmental Toxicology
language:en
Short-container-title:Environ. Toxicol.

Author:

Kao Shang-Jyh¹²,Lee Wei-Jiunn³⁴,Chang Jer-Hwa²⁵,Chow Jyh-Ming⁶,Chung Chi-Li⁷,Hung Wen-Yueh⁸,Chien Ming-Hsien³⁸

Affiliation:

1. Department of Chest Medicine; Shin-Kong Wu Ho-Su Memorial Hospital; Taipei Taiwan

2. School of Respiratory Therapy; College of Medicine, Taipei Medical University; Taipei Taiwan

3. Department of Medical Education and Research; Wan Fang Hospital, Taipei Medical University; Taipei Taiwan

4. Department of Urology, School of Medicine; Taipei Medical University; Taipei Taiwan

5. Division of Pulmonary Medicine; Department of Internal Medicine; Wan Fang Hospital, Taipei Medical University; Taipei Taiwan

6. Division of Hematology and Medical Oncology; Department of Internal Medicine; Wan Fang Hospital, Taipei Medical University; Taipei Taiwan

7. Division of Pulmonary Medicine; Department of Internal Medicine; Taipei Medical University Hospital; Taipei Taiwan

8. Graduate Institute of Clinical Medicine, College of Medicine, Taipei Medical University; Taipei Taiwan

Funder

Shin Kong Wu Ho-Su Memorial Hospital

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Management, Monitoring, Policy and Law,Toxicology,General Medicine

Reference50 articles.

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3. Apoptosis control by death and decoy receptors;Ashkenazi;Curr Opin Cell Biol,1999

4. Treatment of advanced non small cell lung cancer;Bareschino;J Thorac Dis,2011

5. The role of MAPK pathways in the action of chemotherapeutic drugs;Boldt;Carcinogenesis,2002

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