The neurogenic phase of angiotensin II-salt hypertension is prevented by chronic intracerebroventricular administration of benzamil

Author:

Osborn John W.1,Olson Dalay M.1,Guzman Pilar1,Toney Glenn M.2,Fink Gregory D.3

Affiliation:

1. Department of Integrative Biology and Physiology; University of Minnesota; Minneapolis Minnesota

2. Department of Physiology and Center for Biomedical Neuroscience; University of Texas Health Science Center at San Antonio; San Antonio Texas

3. Department of Pharmacology and Toxicology; Michigan State University; East Lansing Michigan

Funder

National Heart, Lung and Blood Institute

Publisher

Wiley

Subject

Physiology (medical),Physiology

Reference34 articles.

1. A role for benzamil-sensitive proteins of the central nervous system in the pathogenesis of salt-dependent hypertension;Abrams;Clin. Exp. Pharmacol. Physiol.,2008

2. Effect of intracerebroventricular benzamil on cardiovascular and central autonomic responses to DOCA-salt treatment;Abrams;Am. J. Physiol.,2010

3. Distribution of epithelial sodium channels and mineralocorticoid receptors in cardiovascular regulatory centers in rat brain;Amin;Am. J. Physiol.,2005

4. A spinal vasopressinergic mechanism mediates hyperosmolality-induced sympathoexciation;Atunes;J. Physiol.,2006

5. Postnatal development of bulk flow in the cerebrospinal fluid of the albino rat: clearance of carboxyl-[14c]inulin after intrathecal infusion;Bass;Brain Res.,1973

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