Inhibition of 3α-hydroxysteroid oxidoreductase and 5α-reductase activity by antiandrogens and indomethacin in the rat prostate
Author:
Publisher
Wiley
Subject
Urology,Oncology
Reference16 articles.
1. Changes in Dihydrotestosterone Metabolism Associated with the Development of Canine Benign Prostatic Hyperplasia*
2. INCREASED RATIO OF 5α-REDUCTASE: 3α(β)-HYDROXYSTEROID DEHYDROGENASE ACTIVITIES IN THE HYPERPLASTIC HUMAN PROSTATE
3. Discrimination between Normal, Hyperplastic and Malignant Human Prostatic Tissues by Enzymatic Profiles
4. 3α-Reduction of 5α-Dihydrotestosterone by Rat Ventral Prostate
5. Partial characterization of the cytosol 3α-hydroxysteroid:NAD(P)+oxidoreductase of rat ventral prostate
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2. Effect of Neurosteroid Modulation on Global Ischaemia-Reperfusion-Induced Cerebral Injury in Mice;The Korean Journal of Physiology & Pharmacology;2013
3. O;Hagers Handbuch der Pharmazeutischen Praxis;1999
4. 3α-Hydroxysteroid oxidoreductase activities in dihydrotestosterone degradation and back-formation in rat prostate and epididymis;The Journal of Steroid Biochemistry and Molecular Biology;1996-06
5. Effects of a new steroidal antiandrogen, TZP-4238 (17 α-acetoxy-6-chloro-2-oxa-4, 6-pregnadiene-3, 20-dione), on spontaneously developed canine benign prostatic hyperplasia;The Prostate;1995-12
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