Chronic high glucose induced INS-1β cell mitochondrial dysfunction: A comparative mitochondrial proteome with SILAC-Reference-Cited by-同舟云学术

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Chronic high glucose induced INS-1β cell mitochondrial dysfunction: A comparative mitochondrial proteome with SILAC

Published:2013-10 Issue:20 Volume:13 Page:3030-3039
ISSN:1615-9853
Container-title:PROTEOMICS
language:en
Short-container-title:Proteomics

Author:

Chen Xiulan¹²,Cui Ziyou¹²,Wei Shasha²³,Hou Junjie¹²,Xie Zhensheng²,Peng Xue²,Li Jing¹²,Cai Tanxi¹²,Hang Haiying¹,Yang Fuquan¹²

Affiliation:

1. Laboratory of Protein and Peptide Pharmaceuticals; Institute of Biophysics; Chinese Academy of Sciences; Beijing China

2. Laboratory of Proteomics; Institute of Biophysics; Chinese Academy of Sciences; Beijing China

3. University of Chinese Academy of Sciences; Beijing China

Publisher

Wiley

Subject

Molecular Biology,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/pmic.201200448/fullpdf

Reference32 articles.

1. The relative contributions of insulin resistance and beta-cell dysfunction to the pathophysiology of type 2 diabetes;Kahn;Diabetologia,2003

2. Beta-cell glucose toxicity, lipotoxicity, and chronic oxidative stress in type 2 diabetes;Robertson;Diabetes,2004

3. Early activation of the fatty acid metabolism pathway by chronic high glucose exposure in rat insulin secretory beta-cells;Coute;Proteomics,2010

4. Modulation of neuronal pentraxin 1 expression in rat pancreatic beta-cells submitted to chronic glucotoxic stress;Schvartz;Mol. Cell. Proteomics,2012

5. Minireview: secondary beta-cell failure in type 2 diabetes-a convergence of glucotoxicity and lipotoxicity;Poitout;Endocrinology,2002

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