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Exposure to BMAA mirrors molecular processes linked to neurodegenerative disease

  • Published:2017-08-24 Issue:17-18 Volume:17 Page:1700161
  • ISSN:1615-9853
  • Container-title:PROTEOMICS
  • language:en
  • Short-container-title:Proteomics

Author:

Beri Joshua1,Nash Tara2,Martin Rubia M.3,Bereman Michael S.123

Affiliation:

1. Department of Chemistry; North Carolina State University; Raleigh NC USA

2. Center for Human Health and the Environment; North Carolina State University; Raleigh NC USA

3. Department of Biological Sciences; North Carolina State University; Raleigh NC USA

Funder

North Carolina State University

Center for Human Health and the Environment

Publisher

Wiley

Subject

Molecular Biology,Biochemistry

Reference58 articles.

Cited by 36 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Activation of retinoid X receptors protects retinal neurons and pigment epithelial cells from BMAA-induced death;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;2024-12

2. Neuronal control of microglia through the mitochondria;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2024-06

3. How does the neurotoxin β-N-methylamino-L-alanine exist in biological matrices and cause toxicity?;Science of The Total Environment;2024-04

4. Evaluation of cyanotoxin L-BMAA effect on α-synuclein and TDP43 proteinopathy;Frontiers in Immunology;2024-03-26

5. Gut Symptoms, Gut Dysbiosis and Gut-Derived Toxins in ALS;International Journal of Molecular Sciences;2024-02-03
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