Acetaminophen Hepatotoxicity: Strong Offense and Weakened Defense-Reference-Cited by-同舟云学术

Acetaminophen Hepatotoxicity: Strong Offense and Weakened Defense

Published:2020-04-29 Issue:5 Volume:71 Page:1530-1532
ISSN:0270-9139
Container-title:Hepatology
language:en
Short-container-title:Hepatology

Author:

Kaplowitz Neil¹,Than Tin Aung¹,Win Sanda¹

Affiliation:

1. University of Southern California Research Center for Liver Disease Division of Gastrointestinal and Liver Disease Keck School of Medicine of USC Los Angeles CA

Publisher

Wiley

Subject

Hepatology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/hep.31189

Reference8 articles.

1. The Nrf2 cell defence pathway: Keap1-dependent and -independent mechanisms of regulation

2. c-Jun N-terminal kinase mediates mouse liver injury through a novel Sab (SH3BP5)-dependent pathway leading to inactivation of intramitochondrial Src

3. c-Jun N-terminal kinase modulates oxidant stress and peroxynitrite formation independent of inducible nitric oxide synthase in acetaminophen hepatotoxicity

4. Novel Therapeutic Approaches Against Acetaminophen-induced Liver Injury and Acute Liver Failure

5. Role of JNK Translocation to Mitochondria Leading to Inhibition of Mitochondria Bioenergetics in Acetaminophen-induced Liver Injury

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1. Ameliorative Effect of Macadamia Nut Protein Peptides on Acetaminophen-Induced Acute Liver Injury in Mice;Journal of Medicinal Food;2024-03-01

2. Targeting innate immune responses to attenuate acetaminophen-induced hepatotoxicity;Biochemical Pharmacology;2022-08

3. Inhibition of p53 Sulfoconjugation Prevents Oxidative Hepatotoxicity and Acute Liver Failure;Gastroenterology;2021-12

4. REPLY:;Hepatology;2020-12-07