Inhibition of oxidative stress and apoptosis by camel milk mitigates cyclosporine‐induced nephrotoxicity: Targeting Nrf2/HO‐1 and AKT/eNOS/NO pathways-Reference-Cited by-同舟云学术

Inhibition of oxidative stress and apoptosis by camel milk mitigates cyclosporine‐induced nephrotoxicity: Targeting Nrf2/HO‐1 and AKT/eNOS/NO pathways

Published:2021-04-05 Issue:6 Volume:9 Page:3177-3190
ISSN:2048-7177
Container-title:Food Science & Nutrition
language:en
Short-container-title:Food Sci Nutr

Author:

Arab Hany H.¹,Eid Ahmed H.²,Gad Amany M.²³,Yahia Rania²,Mahmoud Ayman M.⁴⁵,Kabel Ahmed M.⁶^ORCID

Affiliation:

1. Department of Pharmacology and Toxicology College of Pharmacy Taif University Taif Saudi Arabia

2. Department of Pharmacology Egyptian Drug Authority (EDA), formerly NODCAR Giza Egypt

3. Department of Pharmacology and Toxicology Faculty of Pharmacy Sinai University El Ismailia Egypt

4. Zoology Department, Faculty of Science Beni‐Suef University Beni‐Suef Egypt

5. Biotechnology Department Research Institute of Medicinal and Aromatic Plants Beni‐Suef University Beni‐Suef Egypt

6. Department of Pharmacology Faculty of Medicine Tanta University Tanta Egypt

Funder

Taif University

King Abdulaziz City for Science and Technology

Publisher

Wiley

Subject

Food Science

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/fsn3.2277

Reference41 articles.

1. Camel milk rescues neurotoxic impairments induced by fenpropathrin via regulating oxidative stress, apoptotic, and inflammatory events in the brain of rats

2. Sildenafil protects against nitric oxide deficiency-related nephrotoxicity in cyclosporine A treated rats

3. Activation of autophagy and suppression of apoptosis by dapagliflozin attenuates experimental inflammatory bowel disease in rats: Targeting AMPK/mTOR, HMGB1/RAGE and Nrf2/HO-1 pathways

4. Activation of autophagy by sitagliptin attenuates cadmium-induced testicular impairment in rats: Targeting AMPK/mTOR and Nrf2/HO-1 pathways

5. Mechanistic perspective of morin protection against ketoprofen-induced gastric mucosal injury: Targeting HMGB1/RAGE/NF-κB, DJ-1/Nrf2/HO-1 and PI3K/mTOR pathways

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