Myocardial expression of the anaphylatoxin receptor C3aR is associated with cardiac inflammation and prognosis in patients with non-ischaemic heart failure-Reference-Cited by-同舟云学术

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Myocardial expression of the anaphylatoxin receptor C3aR is associated with cardiac inflammation and prognosis in patients with non-ischaemic heart failure

Published:2018-08-31 Issue:5 Volume:5 Page:846-857
ISSN:2055-5822
Container-title:ESC Heart Failure
language:en
Short-container-title:ESC Heart Failure

Author:

Mueller Karin A.L.¹,Patzelt Johannes¹,Sauter Manuela²,Maier Philipp²,Gekeler Sarah²,Klingel Karin³,Kandolf Reinhard³,Seizer Peter¹,Gawaz Meinrad¹,Geisler Tobias¹,Langer Harald F.¹²

Affiliation:

1. Department of Cardiovascular Medicine; University Hospital, Eberhard Karls University of Tübingen; Tübingen Germany

2. Section for Cardioimmunology; Eberhard Karls University of Tübingen; Tübingen Germany

3. Department for Molecular Pathology; Eberhard Karls University of Tübingen; Tübingen Germany

Funder

TÜFF Frauenförderungsprogramm of the University Tübingen

Deutsche Forschungsgemeinschaft

Volkswagen Foundation

Publisher

Wiley

Subject

Cardiology and Cardiovascular Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/ehf2.12298/fullpdf

Reference42 articles.

1. Contemporary definitions and classification of the cardiomyopathies: an American Heart Association Scientific Statement from the Council on Clinical Cardiology, Heart Failure and Transplantation Committee; Quality of Care and Outcomes Research and Functional Genomics and Translational Biology Interdisciplinary Working Groups; and Council on Epidemiology and Prevention;Maron;Circulation,2006

2. The immune system and chronic heart failure: is the heart in control?;Fildes;J Am Coll Cardiol,2009

3. Immune mechanisms in heart failure;Zhang;Eur J Heart Fail,2017

4. Complement receptor 1 and 2 deficiency increases coxsackievirus B3-induced myocarditis, dilated cardiomyopathy, and heart failure by increasing macrophages, IL-1β, and immune complex deposition in the heart;Fairweather;J Immunol,2006

5. Complement: a key system for immune surveillance and homeostasis;Ricklin;Nat Immunol,2010

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