Murine Placental‐Fetal Phosphate Dyshomeostasis Caused by an Xpr1 Deficiency Accelerates Placental Calcification and Restricts Fetal Growth in Late Gestation-Reference-Cited by-同舟云学术

Murine Placental‐Fetal Phosphate Dyshomeostasis Caused by an Xpr1 Deficiency Accelerates Placental Calcification and Restricts Fetal Growth in Late Gestation

Published:2019-10-25 Issue:1 Volume:35 Page:116-129
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Xu Xuan¹^ORCID,Li Xiunan¹,Sun Hao¹,Cao Zhijian¹,Gao Ruixi¹,Niu Tingting¹,Wang Yanli¹,Ma Tingbin¹,Chen Rui¹,Wang Cheng¹,Yang Zhengang²,Liu Jing Yu¹

Affiliation:

1. Key Laboratory of Molecular Biophysics of the Ministry of Education, College of Life Science and TechnologyHuazhong University of Science and Technology Wuhan China

2. State Key Laboratory of Medical Neurobiology, Department of NeurologyInstitutes of Brain Science, Zhongshan Hospital, Fudan University Shanghai China

Funder

National Key Research and Development Program of China

National Natural Science Foundation of China

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbmr.3866

Reference43 articles.

1. The Phosphate Transporter PiT1 (Slc20a1) Revealed As a New Essential Gene for Mouse Liver Development

2. Slc20a2 deficiency results in fetal growth restriction and placental calcification associated with thickened basement membranes and novel CD13 and lamininα1 expressing cells