Promotion of PTEN on apoptosis through PI3K/Akt signal in vascular smooth muscle cells of mice model of coronary heart disease-Reference-Cited by-同舟云学术

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Promotion of PTEN on apoptosis through PI3K/Akt signal in vascular smooth muscle cells of mice model of coronary heart disease

Published:2019-05-15 Issue:9 Volume:120 Page:14636-14644
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J Cell Biochem

Author:

Wang Sheng¹^ORCID,Cheng Zhaoyun¹,Chen Xianjie¹

Affiliation:

1. Henan Provincial People's Hospital Heart Center Fuwai Central China Cardiovascular Hospital Zhengzhou Henan China

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcb.28725

Reference11 articles.

1. Cardiomyocyte death: mechanisms and translational implications

2. Protection against cerebral infarction by Withaferin A involves inhibition of neuronal apoptosis, activation of PI3K/Akt signaling pathway, and reduced intimal hyperplasia via inhibition of VSMC migration and matrix metalloproteinases

3. Palmitate induces VSMC apoptosis via toll like receptor (TLR)4/ROS/p53 pathway

4. Study of PTEN subcellular localization

5. Phosphatase PTEN is critically involved in post-myocardial infarction remodeling through the Akt/interleukin-10 signaling pathway

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4. Exogenous Hydrogen Sulfide Activates PI3K/Akt/eNOS Pathway to Improve Replicative Senescence in Human Umbilical Vein Endothelial Cells;Cardiology Research and Practice;2023-04-06

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