Changes in antibody titers and clinical course in myasthenia gravis retrospective study
Author:
Affiliation:
1. Dr Berger is a Senior Physician and Dr Schröter is an Assistant Physician at the Clinic of Neurology and Neurophysiology, Medical Center – University of Freiburg, Faculty of Medicine, University of Freiburg, Germany
Publisher
Wiley
Subject
Psychiatry and Mental health,Neurology (clinical),Neurology,Pshychiatric Mental Health
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/pnp.738
Reference41 articles.
1. Myasthenia gravis;Gilhus NE;N Engl J Med,2016
2. Modulation of acetylcholine receptor in rat diaphragm by anti‐receptor sera;Heinemann S;Nature,1978
3. KaoI DrachmanDB.Myasthenic immunoglobulin accelerates acetylcholine receptor degradation.Science1977;196(4289):527–9.
4. MuSK IgG4 autoantibodies cause myasthenia gravis by inhibiting binding between MuSK and Lrp4;Huijbers MG;Proc Natl Acad Sci U S A,2013
5. IgG4 autoantibodies against muscle‐specific kinase undergo Fab‐arm exchange in myasthenia gravis patients;Koneczny I;J Autoimmun,2017
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1. Plasma Exchange versus Intravenous Immunoglobulin in Worsening Myasthenia Gravis: A Systematic Review and Meta-Analysis with Special Attention to Faster Relapse Control;Biomedicines;2023-11-29
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