Differential protein expression in heart in UT-B null mice with cardiac conduction defects-Reference-Cited by-同舟云学术

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Differential protein expression in heart in UT-B null mice with cardiac conduction defects

Published:2009-02 Issue:3 Volume:9 Page:504-511
ISSN:1615-9853
Container-title:PROTEOMICS
language:en
Short-container-title:Proteomics

Author:

Yu Hao,Meng Yan,Wang Li-Shun,Jin Xian,Gao Li-Fang,Zhou Lei,Ji Kun,Li Yang,Zhao Li-Juan,Chen Guo-Qiang,Zhao Xue-Jian,Yang Baoxue

Publisher

Wiley

Subject

Molecular Biology,Biochemistry

Reference37 articles.

1. Identification of a gene responsible for familial Wolff-Parkinson-White syndrome;Gollob;New Engl. J. Med.,2001

2. Novel PRKAG2 mutation responsible for the genetic syndrome of ventricular preexcitation and conduction system disease with childhood onset and absence of cardiac hypertrophy;Gollob;Circulation,2001

3. Recognition and management of fatty acid oxidation defects: a series of 107 patients;Saudubray;J. Inherit. Metab. Dis.,1999

4. Syntrophin gamma 2 regulates SCN5A gating by a PDZ domain-mediated interaction;Ou;J. Biol. Chem.,2003

5. A mutation in the human cardiac sodium channel (E161K) contributes to sick sinus syndrome, conduction disease and Brugada syndrome in two families;Smits;J. Mol. Cell. Cardiol.,2005

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3. Physiological functions of urea transporter B;Pflügers Archiv - European Journal of Physiology;2019-11-22

4. Urea transporter proteins as targets for small-molecule diuretics;Nature Reviews Nephrology;2014-12-09

5. Quantitative proteomic study of myocardial mitochondria in urea transporter B knockout mice;PROTEOMICS;2014-08-08

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