Subcellular organization of camkii in rat hippocampal pyramidal neurons-Reference-Cited by-同舟云学术

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Subcellular organization of camkii in rat hippocampal pyramidal neurons

Published:2013-08-23 Issue:15 Volume:521 Page:3570-3583
ISSN:0021-9967
Container-title:Journal of Comparative Neurology
language:en
Short-container-title:J. Comp. Neurol.

Author:

Ding Jin-Dong¹,Kennedy Mary B.²,Weinberg Richard J.³

Affiliation:

1. Department of Ophthalmology; Duke University Medical Center; Durham North Carolina 27710

2. Department of Biology; California Institute of Technology; Pasadena California 91125

3. Department of Cell Biology and Physiology, and Neuroscience Center; University of North Carolina; Chapel Hill North Carolina 27599

Publisher

Wiley

Subject

General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/cne.23372/fullpdf

Reference53 articles.

1. Ischemia-induced translocation of Ca2+/calmodulin-dependent protein kinase II: potential role in neuronal damage;Aronowski;J Neurochem,1992

2. NMDA receptor subunit composition controls synaptic plasticity by regulating binding to CaMKII;Barria;Neuron,2005

3. Regulatory phosphorylation of AMPA-type glutamate receptors by CaM-KII during long-term potentiation;Barria;Science,1997

4. The metabotropic glutamate receptor (mGluR1 α) is concentrated at perisynaptic membrane of neuronal subpopulations as detected by immunogold reaction;Baude;Neuron,1993

5. Developmental expression of the CaM kinase II isoforms: ubiquitous γ- and δ-CaM kinase II are the early isoforms and most abundant in the developing nervous system;Bayer;Brain Res Mol Brain Res,1999

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4. Spatiotemporal modelling reveals geometric dependence of AMPAR dynamics on dendritic spine morphology;The Journal of Physiology;2022-11-23

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