RELA is sufficient to mediate interleukin‐1 repression of androgen receptor expression and activity in an LNCaP disease progression model-Reference-Cited by-同舟云学术

RELA is sufficient to mediate interleukin‐1 repression of androgen receptor expression and activity in an LNCaP disease progression model

Published:2019-11-15 Issue:2 Volume:80 Page:133-145
ISSN:0270-4137
Container-title:The Prostate
language:en
Short-container-title:Prostate

Author:

Thomas‐Jardin Shayna E.¹,Dahl Haley¹,Kanchwala Mohammed S.²,Ha Freedom¹,Jacob Joan¹,Soundharrajan Reshma¹,Bautista Monica¹,Nawas Afshan F.¹,Robichaux Dexter¹,Mistry Ragini¹,Anunobi Vanessa¹,Xing Chao²³⁴,Delk Nikki A.¹^ORCID

Affiliation:

1. Biological Sciences DepartmentThe University of Texas at DallasRichardson Texas

2. McDermott Center of Human Growth and DevelopmentThe University of Texas Southwestern Medical CenterDallas Texas

3. Department of BioinformaticsThe University of Texas Southwestern Medical CenterDallas Texas

4. Department of Clinical SciencesThe University of Texas Southwestern Medical CenterDallas Texas

Funder

National Institutes of Health

National Cancer Institute

University of Texas System

Publisher

Wiley

Subject

Urology,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/pros.23925

Reference29 articles.

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3. Interleukin 1β mediates the modulatory effects of monocytes on LNCaP human prostate cancer cells

4. IL-1β Inducesp62/SQSTM1and RepressesAndrogen ReceptorExpression in Prostate Cancer Cells

5. Anti‐androgen resistance in prostate cancer cells chronically induced by interleukin‐1β;Staverosky JA;Am J Clin Exp Urol,2013

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