The transcription factor FBI ‐1 inhibits SAM 68‐mediated BCL ‐ X alternative splicing and apoptosis-Reference-Cited by-同舟云学术

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The transcription factor FBI ‐1 inhibits SAM 68‐mediated BCL ‐ X alternative splicing and apoptosis

Published:2014-02-10 Issue:4 Volume:15 Page:419-427
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Rep

Author:

Bielli Pamela¹²,Busà Roberta¹²,Di Stasi Savino M³,Munoz Manuel J⁴,Botti Flavia¹,Kornblihtt Alberto R⁴,Sette Claudio¹²

Affiliation:

1. Department of Biomedicine and Prevention University of Rome Tor Vergata Rome Italy

2. Laboratory of Neuroembryology Fondazione Santa Lucia Rome Italy

3. Department of Experimental Medicine and Surgery University of Rome Tor Vergata Rome Italy

4. Laboratorio de Fisiologia y Biologia Molecular Universidad de Buenos Aires Buenos Aires Argentina

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/embr.201338241

Reference26 articles.

1. Mechanisms of alternative splicing regulation: insights from molecular and genomics approaches

2. Functional consequences of developmentally regulated alternative splicing

3. The Spliceosome: Design Principles of a Dynamic RNP Machine

4. Mechanisms of Alternative Pre-Messenger RNA Splicing

5. Regulation of Apoptosis by Alternative Pre-mRNA Splicing

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2. An in vitro CRISPR screen of cell-free DNA identifies apoptosis as the primary mediator of cell-free DNA release;Communications Biology;2024-04-10

3. Comprehensive Atlas of Alternative Splicing Reveals NSRP1 Promoting Adipogenesis through CCDC18;International Journal of Molecular Sciences;2024-03-01

4. Differential expression of paralog RNA binding proteins establishes a dynamic splicing program required for normal cerebral cortex development;Nucleic Acids Research;2024-02-07

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