Ginsenoside‐Rg1 attenuates sepsis‐induced cardiac dysfunction by modulating mitochondrial damage via the P2X7 receptor‐mediated Akt/GSK‐3β signaling pathway-Reference-Cited by-同舟云学术

Ginsenoside‐Rg1 attenuates sepsis‐induced cardiac dysfunction by modulating mitochondrial damage via the P2X7 receptor‐mediated Akt/GSK‐3β signaling pathway

Published:2021-12-02 Issue:1 Volume:36 Page:
ISSN:1095-6670
Container-title:Journal of Biochemical and Molecular Toxicology
language:en
Short-container-title:J Biochem & Molecular Tox

Author:

Liu Zhengyu¹²,Pan Hongwei¹²,Zhang Yixiong³,Zheng Zhaofen¹²,Xiao Weiwei³,Hong Xiuqin⁴,Chen Fang³,Peng Xiang²,Pei Yanfang³,Rong Jingjing¹²,He Jin¹²,Zou Lianhong⁵,Wang Jia⁴,Zhong Jie⁴,Han Xiaotong³^ORCID,Cao Yan³^ORCID

Affiliation:

1. Cardiology Department Hunan Provincial People's Hospital (The First Affiliated Hospital of Hunan Normal University) Changsha China

2. Clinical Medicine Research Center of Heart Failure of Hunan Province Hunan Provincial People's Hospital (The First Affiliated Hospital of Hunan Normal University) Changsha China

3. Emergency Department Hunan Provincial People's Hospital (The First Affiliated Hospital of Hunan Normal University) Changsha China

4. Department of Research Hunan Provincial People's Hospital (The First Affiliated Hospital of Hunan Normal University) Changsha China

5. Hunan Provincial Key Laboratory of Emergency and Critical Care Metabolomics Hunan Provincial People's Hospital (The First Affiliated Hospital of Hunan Normal University) Changsha China

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Toxicology,Molecular Biology,Molecular Medicine,Biochemistry,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbt.22885

Reference33 articles.

1. The Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3)

2. The Pathogenesis of Sepsis and Potential Therapeutic Targets

3. Surviving Sepsis Campaign: International Guidelines for Management of Sepsis and Septic Shock: 2016

4. Role of Oxidative Stress and Mitochondrial Dysfunction in Sepsis and Potential Therapies

5. Mechanisms of sepsis-induced cardiac dysfunction

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