Decrease in cellularity and expression of adhesion molecules by anti–tumor necrosis factor α monoclonal antibody treatment in patients with rheumatoid arthritis-Reference-Cited by-同舟云学术

Decrease in cellularity and expression of adhesion molecules by anti–tumor necrosis factor α monoclonal antibody treatment in patients with rheumatoid arthritis

Published:1996-07 Issue:7 Volume:39 Page:1077-1081
ISSN:0004-3591
Container-title:Arthritis & Rheumatism
language:en
Short-container-title:Arthritis & Rheumatism

Author:

Tak Paul P.,Taylor Peter C.,Breedveld Ferdinand C.,Smeets Tom J. M.,Daha Mohamed R.,Kluin Philip M.,Meinders A. Edo,Maini Ravinder N.

Publisher

Wiley

Subject

Pharmacology (medical),Immunology,Rheumatology,Immunology and Allergy

Reference17 articles.

1. TNFα—A PIVOTAL ROLE IN RHEUMATOID ARTHRITIS?

2. Monoclonal anti-TNFalpha Antibody as a Probe of Pathogenesis and Therapy of Rheumatoid Disease

3. Treatment of rheumatoid arthritis with chimeric monoclonal antibodies to tumor necrosis factor α

4. Randomised double-blind comparison of chimeric monoclonal antibody to tumour necrosis factor α (cA2) versus placebo in rheumatoid arthritis

5. The american rheumatism association 1987 revised criteria for the classification of rheumatoid arthritis

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