Neuroprotection against permanent focal cerebral ischemia by ginkgolides A and B is associated with obstruction of the mitochondrial apoptotic pathway via inhibition of c‐Jun N‐terminal kinase in rats
Author:
Affiliation:
1. Department of PharmacyPutuo People's HospitalShanghai China
2. Department of Clinical LaboratoryTongji Hospital of Tongji UniversityShanghai China
3. Institute of PathologyCharité University HospitalBerlin Germany
Funder
National Natural Science Foundation of China
Shanghai Pujiang Program
Research Program of the Department of Healthcare of Putuo District, Shanghai
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jnr.23306
Reference50 articles.
1. The antimicrobial peptide LL-37 modulates the inflammatory and host defense response of human neutrophils
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3. Amino-terminal phosphorylation of c-Jun regulates stress-induced apoptosis and cellular proliferation
4. JNK Signalling: A Possible Target to Prevent Neurodegeneration
5. A peptide inhibitor of c-Jun N-terminal kinase protects against excitotoxicity and cerebral ischemia
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