Effect of serial cell passaging in the retention of fiber type and mitochondrial content in primary human myotubes-Reference-Cited by-同舟云学术

Effect of serial cell passaging in the retention of fiber type and mitochondrial content in primary human myotubes

Published:2015-11-05 Issue:12 Volume:23 Page:2414-2420
ISSN:1930-7381
Container-title:Obesity
language:en
Short-container-title:Obesity

Author:

Covington Jeffrey D.¹²,Myland Cassandra K.¹,Rustan Arild C.³,Ravussin Eric¹,Smith Steven R.⁴,Bajpeyi Sudip¹⁵

Affiliation:

1. Laboratory of Skeletal Muscle Physiology; Pennington Biomedical Research Center; Baton Rouge Louisiana USA

2. School of Medicine, Louisiana State University Health Sciences Center; New Orleans Louisiana USA

3. Department of Pharmaceutical Biosciences; School of Pharmacy, University of Oslo; Oslo Norway

4. Translational Research Institute for Metabolism and Diabetes; Florida Hospital, Sanford-Burnham Medical Research Institute; Winter Park Florida USA

5. Department of Kinesiology; University of Texas at El Paso; El Paso Texas USA

Funder

Novartis, Novartis Clinical Innovation Fund

Takeda Pharmaceuticals North America

NIH

NORC Center

NIDDK and COBRE

COBRE

NORC

Publisher

Wiley

Subject

Nutrition and Dietetics,Endocrinology,Endocrinology, Diabetes and Metabolism,Medicine (miscellaneous)

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/oby.21192/fullpdf

Reference37 articles.

1. Reduced lipid oxidation in myotubes established from obese and type 2 diabetic subjects;Gaster;Biochem Biophys Res Commun,2009

2. Mitochondrial respiration is decreased in skeletal muscle of patients with type 2 diabetes;Mogensen;Diabetes,2007

3. Skeletal muscle mitochondrial capacity and insulin resistance in type 2 diabetes;Bajpeyi;J Clin Endocrinol Metab,2011

4. Intramyocellular lipid concentrations are correlated with insulin sensitivity in humans: a 1H NMR spectroscopy study;Krssak;Diabetologia,1999

5. Nine months of combined training improves ex vivo skeletal muscle metabolism in individuals with type 2 diabetes;Sparks;J Clin Endocrinol Metab,2013

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