Aggravated intestinal apoptosis by ClC-3 deletion is lethal to mice endotoxemia-Reference-Cited by-同舟云学术

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Aggravated intestinal apoptosis by ClC-3 deletion is lethal to mice endotoxemia

Published:2018-08-10 Issue:10 Volume:42 Page:1445-1453
ISSN:1065-6995
Container-title:Cell Biology International
language:en
Short-container-title:Cell Biol Int

Author:

Huang Lin-Yan¹,Li Yu-Jie¹,Li Peng-Peng²,Li Hong-Chun¹,Ma Ping¹²

Affiliation:

1. School of Medical Technology; Xuzhou Medical University; 209 Tongshan Rd Xuzhou Jiangsu 221004 P.R. China

2. Department of Medical Laboratory; The Affiliated Hospital of Xuzhou Medical University; No.99 Huaihai West Road Xuzhou Jiangsu 221000 P.R. China

Funder

China Postdoctoral Science Foundation

Natural Science Foundation of Jiangsu Province

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,General Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/cbin.11025/fullpdf

Reference25 articles.

1. The two faces of IKK and NF-kappaB inhibition: prevention of systemic inflammation but increased local injury following intestinal ischemia-reperfusion;Chen;Nat Med,2003

2. A critical role for chloride channel-3 (CIC-3) in smooth muscle cell activation and neointima formation;Chu;Arteriosclerosis, Thromb Vascular Biol,2011

3. Inhibition of bcl-2 as cancer therapy;Ciardiello;Ann Oncol,2002

4. Role of lipopolysaccharide and cecal ligation and puncture on blood coagulation and inflammation in sensitive and resistant mice models;Corral;Am J Pathol,2005

5. Molecular identification of a volume-regulated chloride channel;Duan;Nature,1997

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3. ClC-3: A Novel Promising Therapeutic Target for Atherosclerosis;Journal of Cardiovascular Pharmacology and Therapeutics;2021-06-17

4. 17β-Estradiol activates Cl− channels via the estrogen receptor α pathway in human thyroid cells;Channels;2021-01-01

5. Suppression of CLC-3 reduces the proliferation, invasion and migration of colorectal cancer through Wnt/β-catenin signaling pathway;Biochemical and Biophysical Research Communications;2020-12

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