Mechanism of pancreatic and liver malformations in human fetuses with short-rib polydactyly syndrome-Reference-Cited by-同舟云学术

Mechanism of pancreatic and liver malformations in human fetuses with short-rib polydactyly syndrome

Published:2016-03-11 Issue:7 Volume:106 Page:549-562
ISSN:1542-0752
Container-title:Birth Defects Research Part A: Clinical and Molecular Teratology
language:en
Short-container-title:Birth Defects Research Part A: Clinical and Molecular Teratology

Author:

Loo Christine K.C.¹²³,Pereira Tamara N.²,Ramsing Mette⁴,Vogel Ida⁵,Petersen Olav B.⁶,Ramm Grant A.²⁷

Affiliation:

1. Department of Anatomical Pathology SEALS; Prince of Wales Hospital; Sydney Australia (formerly: Department of Anatomical Pathology, Royal Brisbane and Women's Hospital, Brisbane, Australia.)

2. Hepatic Fibrosis Group; QIMR Berghofer Medical Research Institute; Brisbane Australia

3. Discipline of Pathology School of Medicine; University of Western Sydney; Australia

4. Department of Pathology; Aarhus University Hospital; Denmark

5. Department of Clinical Genetics; Aarhus University Hospital; Denmark

6. Department of Obstetrics and Gynaecology; Aarhus University Hospital; Denmark

7. Faculty of Medicine and Biomedical Sciences; The University of Queensland; Brisbane Australia

Funder

The National Health and Medical Research Council (NHMRC) of Australia

The Study, Education, Research Trust Fund (SERTF)

Pathology Queensland

The NHMRC of Australia

Publisher

Wiley

Subject

Developmental Biology,Embryology,General Medicine,Pediatrics, Perinatology, and Child Health

Reference45 articles.

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5. Stromal cell-derived factor 1 (SDF-1) and antenatal human B cell lymphopoiesis: expression of SDF-1 by mesothelial cells and biliary ductal plate epithelial cells;Coulomb-L'Hermin;Proc Natl Acad Sci U S A,1999

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