Integration of amplifiedBCR/ABL fusion genes into the short arm of chromosome 17 as a novel mechanism of disease progression in chronic myeloid leukemia-Reference-Cited by-同舟云学术

Integration of amplifiedBCR/ABL fusion genes into the short arm of chromosome 17 as a novel mechanism of disease progression in chronic myeloid leukemia

Published:2001 Issue:1 Volume:31 Page:10-14
ISSN:1045-2257
Container-title:Genes, Chromosomes and Cancer
language:en
Short-container-title:Genes Chromosom. Cancer

Author:

Metzke-Heidemann Simone,Harder Lana,Gesk Stefan,Schoch Robert,Jenisch Stefan,Grote Werner,Siebert Reiner,Schlegelberger Brigitte

Publisher

Wiley

Subject

Cancer Research,Genetics

Reference20 articles.

1. C-abl and bcr are rearranged in a Ph1-negative CML patient.

2. The c-abl, bcr and cλ genes are amplified in a cell line but not in the uncultured cells from a patient with chronic myelogenous leukemia

3. High incidence of a second BCR-ABL fusion in chronic myeloid leukemia revealed by interphase cytogenetic analysis on blood and bone marrow smears

4. A study of prognostic factors in blast crisis of Philadelphia chromosome-positive chronic myelogenous leukaemia

5. Translocation ofBCR to chromosome 9: A new cytogenetic variant detected by FISH in two Ph-negative,BCR-positive patients with chronic myeloid leukemia

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3. Angiopoietin-2 predicts disease-free survival after allogeneic stem cell transplantation in patients with high-risk myeloid malignancies;Blood;2008-09-01

4. The short arm of chromosome 18 is a recurrent integration site of a supernumerary BCR/ABL gene fusion in chronic myeloid leukemia;Cancer Genetics and Cytogenetics;2007-12

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