Antisense suppression of glial fibrillary acidic protein as a treatment for Alexander disease
Author:
Affiliation:
1. Waisman Center; University of Wisconsin-Madison; Madison WI
2. Ionis Pharmaceuticals; Carlsbad CA
3. Department of Comparative Biosciences; University of Wisconsin-Madison; Madison WI
Funder
NIH
United Leukodystrophy Foundation
Juanma Fund
Publisher
Wiley
Subject
Neurology (clinical),Neurology
Reference47 articles.
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2. Alexander disease;Messing;J Neurosci,2012
3. IF-pathies”: a broad spectrum of intermediate filament-associated diseases;Omary;J Clin Invest,2009
4. Mice lacking glial fibrillary acidic protein display astrocytes devoid of intermediate filaments but develop and reproduce normally;Pekny;EMBO J,1995
5. Targeted deletion in astrocyte intermediate filament (Gfap) alters neuronal physiology;McCall;Proc Natl Acad Sci U S A,1996
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