Myosin VI and cardiomyopathy: Left ventricular hypertrophy, fibrosis, and both cardiac and pulmonary vascular endothelial cell defects in the Snell's waltzer mouse-Reference-Cited by-同舟云学术

Myosin VI and cardiomyopathy: Left ventricular hypertrophy, fibrosis, and both cardiac and pulmonary vascular endothelial cell defects in the Snell's waltzer mouse

Published:2015-08 Issue:8 Volume:72 Page:373-387
ISSN:1949-3584
Container-title:Cytoskeleton
language:en
Short-container-title:Cytoskeleton

Author:

Hegan Peter S.¹,Lanahan Anthony A.²,Simons Michael²³,Mooseker Mark S.¹³⁴

Affiliation:

1. Department of Molecular, Cellular and Developmental Biology; Yale University; New Haven Connecticut

2. Department of Internal Medicine; Yale School of Medicine; New Haven Connecticut

3. Department of Cell Biology; Yale School of Medicine; New Haven Connecticut

4. Department of Pathology; Yale School of Medicine; New Haven Connecticut

Funder

NIH

Publisher

Wiley

Subject

Cell Biology,Structural Biology

Reference38 articles.

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2. Defective CFTR apical endocytosis and enterocyte brush border in myosin VI-deficient mice;Ameen;Traffic,2007

3. The mouse Snell's waltzer deafness gene encodes an unconventional myosin required for structural integrity of inner ear hair cells;Avraham;Nat Genet,1995

4. Multifunctional myosin VI has a multitude of cargoes;Buss;Proc Natl Acad Sci USA,2011

5. Transgenic mouse models in angiogenesis and cardiovascular disease;Carmeliet;J Pathol,2000

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