17β-estradiol reduces Cav1.2 channel abundance and attenuates Ca2+-dependent contractions in coronary arteries

Author:

Hill Brent J.F.1ORCID,Dalton Robin J.1,Joseph Biny K.2,Thakali Keshari M.3,Rusch Nancy J.2

Affiliation:

1. Department of Biology; University of Central Arkansas Conway; Conway Arkansas

2. Department of Pharmacology and Toxicology; College of Medicine; University of Arkansas for Medical Sciences; Little Rock Arkansas

3. Arkansas Children's Nutrition Center & Department of Pediatrics; University of Arkansas for Medical Sciences; Little Rock Arkansas

Funder

Arkansas INBRE program

National Institutes of Health

National Institute of General Medical Sciences

Publisher

Wiley

Subject

General Pharmacology, Toxicology and Pharmaceutics,Neurology

Reference62 articles.

1. The Cavβ subunit prevents RFP2-mediated ubiquitination and proteasomal degradation of L-type channels;Altier;Nat Neurosci,2011

2. Gender differences in endothelium-dependent relaxations do not involve NO in porcine coronary arteries;Barber;Am J Physiol,1997

3. Gender influences coronary L-type Ca2+ current and adaptation to exercise training in miniature swine;Bowles;J Appl Physiol,2001

4. Exercise training increases L-type calcium current density in coronary smooth muscle;Bowles;Am J Physiol,1998a

5. Exercise training increases K+-channel contribution to regulation of coronary arterial tone;Bowles;J Appl Physiol,1998b

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