A rat model of severe VWD by elimination of the VWF gene using CRISPR/Cas9-Reference-Cited by-同舟云学术

A rat model of severe VWD by elimination of the VWF gene using CRISPR/Cas9

Published:2019-12-29 Issue:1 Volume:4 Page:64-71
ISSN:2475-0379
Container-title:Research and Practice in Thrombosis and Haemostasis
language:en
Short-container-title:Res Pract Thromb Haemost

Author:

Garcia Jessica¹,Flood Veronica H.²³,Haberichter Sandra L.³,Fahs Scot A.³,Mattson Jeremy G.³,Geurts Aron M.³,Zogg Mark⁴,Weiler Hartmut³,Shi Qizhen²³,Montgomery Robert R.²³

Affiliation:

1. Department of Pediatrics Division of Hematology/Oncology UT Southwestern Medical Center Dallas TX USA

2. Department of Pediatrics Division of Hematology/Oncology Medical College of Wisconsin Milwaukee WI USA

3. Blood Research Institute BloodCenter of Wisconsin Milwaukee WI USA

4. Department of Physiology Medical College of Wisconsin Milwaukee WI USA

Funder

National Hemophilia Foundation

National Institutes of Health

Publisher

Wiley

Subject

Hematology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/rth2.12280

Reference22 articles.

1. von Willebrand disease (VWD): evidence-based diagnosis and management guidelines, the National Heart, Lung, and Blood Institute (NHLBI) Expert Panel report (USA)

2. Clinical manifestations and complications of childbirth and replacement therapy in 385 Iranian patients with type 3 von Willebrand disease

3. BIOCHEMISTRY AND GENETICS OF VON WILLEBRAND FACTOR

4. Isolation and characterization of two domains of human von Willebrand factor that interact with fibrillar collagen types I and III.

5. Crucial role for the VWF A1 domain in binding to type IV collagen

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1. A novel mouse model of type 2N VWD was developed by CRISPR/Cas9 gene editing and recapitulates human type 2N VWD;Blood Advances;2022-05-02

2. Advances in Genome Editing and Application to the Generation of Genetically Modified Rat Models;Frontiers in Genetics;2021-04-20