Mitochondrial ROS control neuronal excitability and cell fate in frontotemporal dementia-Reference-Cited by-同舟云学术

Mitochondrial ROS control neuronal excitability and cell fate in frontotemporal dementia

Published:2021-05-31 Issue: Volume: Page:
ISSN:1552-5260
Container-title:Alzheimer's & Dementia
language:en
Short-container-title:Alzheimer's & Dementia

Author:

Esteras Noemí¹^ORCID,Kopach Olga²,Maiolino Marta³,Lariccia Vincenzo³,Amoroso Salvatore³,Qamar Seema⁴,Wray Selina⁵,Rusakov Dmitri A.²,Jaganjac Morana⁶⁷,Abramov Andrey Y.¹

Affiliation:

1. Department of Clinical and Movement Neurosciences UCL Queen Square Institute of Neurology London UK

2. Department of Clinical and Experimental Epilepsy UCL Queen Square Institute of Neurology London UK

3. Department of Biomedical Sciences and Public Health School of Medicine University “Politecnica delle Marche,” Ancona Italy

4. Department of Clinical Neurosciences Cambridge Institute for Medical Research University of Cambridge Cambridge UK

5. Department of Neurodegenerative Disease UCL Queen Square Institute of Neurology London UK

6. Qatar Analytics & BioResearch Lab Anti‐Doping Lab Qatar Doha Qatar

7. Division of Molecular Medicine Rudjer Boskovic Institute Zagreb Croatia

Publisher

Wiley

Subject

Psychiatry and Mental health,Cellular and Molecular Neuroscience,Geriatrics and Gerontology,Neurology (clinical),Developmental Neuroscience,Health Policy,Epidemiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/alz.12394

Reference76 articles.

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4. Calcium dysregulation contributes to neurodegeneration in FTLD patient iPSC-derived neurons

5. Tau inhibits mitochondrial calcium efflux and makes neurons vulnerable to calcium-induced cell death

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