Impaired recognition of apoptotic neutrophils by the C1q/calreticulin and CD91 pathway in systemic lupus erythematosus-Reference-Cited by-同舟云学术

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Impaired recognition of apoptotic neutrophils by the C1q/calreticulin and CD91 pathway in systemic lupus erythematosus

Published:2006 Issue:5 Volume:54 Page:1543-1556
ISSN:0004-3591
Container-title:Arthritis & Rheumatism
language:en
Short-container-title:Arthritis Rheum

Author:

Donnelly Suzanne,Roake Wendy,Brown Simon,Young Philip,Naik Haley,Wordsworth Paul,Isenberg David A.,Reid Kenneth B. M.,Eggleton Paul

Publisher

Wiley

Subject

Pharmacology (medical),Immunology,Rheumatology,Immunology and Allergy

Reference53 articles.

1. C1q and Systemic Lupus Erythematosus

2. C1q Knock-Out Mice for the Study of Complement Deficiency in Autoimmune Disease

3. Altered (Oxidized) C1q Induces a Rheumatoid Arthritis- like Destructive and Chronic Inflammation in Joint Structures in Arthritis-Susceptible Rats

4. Common silent mutations in all types of hereditary complement C1q deficiencies

5. Homozygous single nucleotide polymorphism of the complement C1QA gene is associated with decreased levels of C1q in patients with subacute cutaneous lupus erythematosus

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