NOXA‐mediated degradation of MCL1 and BCL2L1 causes apoptosis of daunorubicin‐treated human acute myeloid leukemia cells-Reference-Cited by-同舟云学术

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NOXA‐mediated degradation of MCL1 and BCL2L1 causes apoptosis of daunorubicin‐treated human acute myeloid leukemia cells

Published:2021-05-13 Issue:11 Volume:236 Page:7356-7375
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Chiou Jing‐Ting¹,Huang Nan‐Chieh²,Huang Chia‐Hui¹,Wang Liang‐Jun¹,Lee Yuan‐Chin¹,Shi Yi‐Jun¹,Chang Long‐Sen¹³^ORCID

Affiliation:

1. Institute of Biomedical Sciences National Sun Yat‐Sen University Kaohsiung Taiwan

2. Department of Family Medicine Zuoying Branched of Kaohsiung Armed Forces General Hospital Kaohsiung Taiwan

3. Department of Biotechnology Kaohsiung Medical University Kaohsiung Taiwan

Funder

Ministry of Science and Technology, Taiwan

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.30407

Reference55 articles.

1. Gene and protein analysis reveals that p53 pathway is functionally inactivated in cytogenetically normal Acute Myeloid Leukemia and Acute Promyelocytic Leukemia

2. Intercalation of antitumor drug doxorubicin and its analogue by DNA duplex: Structural features and biological implications

3. Time dependent response of daunorubicin on cytotoxicity, cell cycle and DNA repair in acute lymphoblastic leukaemia

4. Targeting BCL2 for the Treatment of Lymphoid Malignancies

5. Beneficial effects of angiotensin II receptor blocker, olmesartan, in limiting the cardiotoxic effect of daunorubicin in rats

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