Parkinson's Disease, the Subthalamic Nucleus, Inhibition, and Impulsivity

Author:

Jahanshahi Marjan1,Obeso Ignacio2,Baunez Christelle3,Alegre Manuel4,Krack Paul5

Affiliation:

1. Cognitive Motor Neuroscience Group and Unit of Functional Neurosurgery, Sobell Department of Motor Neuroscience and Movement Disorders, UCL Institute of Neurology; London United Kingdom

2. CINAC, HM-Puerta del Sur, Hospitales de Madrid; CEU-San Pablo University, Móstoles; Madrid Spain

3. Basal Ganglia, Motivation and Reward' (BAGAMORE), Institut de Neurosciences de la Timone, UMR7289 CNRS and AMU (Aix Marseille Universite); Marseille France

4. Neurophysiology Laboratory, Neuroscience Area, CIMA, University of Navarra; Pamplona Spain

5. INSERM U836, F-38000 Grenoble, France; University Grenoble Alpes, GIN, Grenoble, France, and CHU de Grenoble, Movement Disorder Unit; Grenoble France

Publisher

Wiley

Subject

Clinical Neurology,Neurology

Reference137 articles.

1. The functional anatomy of basal ganglia disorders;Albin;Trends Neurosci,1989

2. Primate models of movement disorders of basal ganglia origin;DeLong;Trends Neurosci,1990

3. The factors that induce or overcome freezing of gait in Parkinson's disease;Rahman;Behav Neurol,2008

4. Pathophysiology of levodopa-induced dyskinesias in Parkinson's disease: problems with the current model;Obeso;Ann Neurol,2000

5. Executive dysfunction in Parkinson's disease: a review;Dirnberger;J Neuropsychol,2013

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