Injury and differentiation following inhibition of mitochondrial respiratory chain complex IV in rat oligodendrocytes-Reference-Cited by-同舟云学术

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Injury and differentiation following inhibition of mitochondrial respiratory chain complex IV in rat oligodendrocytes

Published:2010-07-21 Issue:15 Volume:58 Page:1827-1837
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Ziabreva Iryna,Campbell Graham,Rist Julia,Zambonin Jessica,Rorbach Joanna,Wydro Mateusz M.,Lassmann Hans,Franklin Robin J. M.,Mahad Don

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/glia.21052/fullpdf

Reference60 articles.

1. Preferential loss of myelin-associated glycoprotein reflects hypoxia-like white matter damage in stroke and inflammatory brain diseases;Aboul-Enein;J Neuropathol Exp Neurol,2003

2. Production of reactive oxygen species in brain mitochondria: Contribution by electron transport chain and non-electron transport chain sources;Adam-Vizi;Antioxid Redox Signal,2005

3. Exposure of developing oligodendrocytes to cadmium causes HSP72 induction, free radical generation, reduction in glutathione levels, and cell death;Almazan;Free Radic Biol Med,2000

4. TNFalpha potentiates IFNgamma-induced cell death in oligodendrocyte progenitors;Andrews;J Neurosci Res,1998

5. Maturation-dependent vulnerability of oligodendrocytes to oxidative stress-induced death caused by glutathione depletion;Back;J Neurosci,1998

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