Inhibition of endoplasmic reticulum stress and the downstream pathways protects CD4 + T cells against apoptosis and immune dysregulation in sepsis
Author:
Affiliation:
1. Department of Trauma Emergency & Critical Care Medicine, Shanghai Fifth People's Hospital Fudan University Shanghai China
2. Department of Geriatrics, Shanghai Fifth People's Hospital Fudan University Shanghai China
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Genetics,Molecular Biology,Biochemistry
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/iub.2666
Reference44 articles.
1. The Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3)
2. Genome-wide recessive genetic screening in mammalian cells with a lentiviral CRISPR-guide RNA library
3. Endoplasmic reticulum stress induces calcium-dependent permeability transition, mitochondrial outer membrane permeabilization and apoptosis
4. Endotoxin causes functional endoplasmic reticulum failure, possibly mediated by mitochondria
5. Endoplasmic reticulum stress in health and disease
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