Cathepsin B but not cathepsins L or S contributes to the pathogenesis of Unverricht-Lundborg progressive myoclonus epilepsy (EPM1)-Reference-Cited by-同舟云学术

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Cathepsin B but not cathepsins L or S contributes to the pathogenesis of Unverricht-Lundborg progressive myoclonus epilepsy (EPM1)

Published:2003-08-08 Issue:4 Volume:56 Page:315-327
ISSN:0022-3034
Container-title:Journal of Neurobiology
language:en
Short-container-title:J. Neurobiol.

Author:

Houseweart Megan K.,Pennacchio Len A.,Vilaythong Alex,Peters Christoph,Noebels Jeffrey L.,Myers Richard M.

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,General Neuroscience

Reference76 articles.

1. Apoptosis induced by exposure to a low steady-state concentration of H2O2 is a consequence of lysosomal rupture

2. Regionally selective changes in brain lysosomes occur in the transition from young adulthood to middle age in rats

3. The human brm protein is cleaved during apoptosis: The role of cathepsin G

4. Photo-Oxidative Disruption of Lysosomal Membranes Causes Apoptosis of Cultured Human Fibroblasts

5. Cytochemical evidence for the leakage of acid phosphatase through ultrastructurally intact lysosomal membranes

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1. Challenges in Batch-to-Bed Translation Involving Inflammation-Targeting Compounds in Chronic Epilepsy: The Case of Cathepsin Activity-Based Probes;ACS Omega;2024-02-01

2. In depth behavioral phenotyping unravels complex motor disturbances in Cstb−/− mouse, a model for progressive myoclonus epilepsy type 1;Frontiers in Behavioral Neuroscience;2023-12-21

3. Progressive mitochondrial dysfunction in cerebellar synaptosomes of cystatin B-deficient mice;Frontiers in Molecular Neuroscience;2023-05-12

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5. Insights into the Genetic Profile of Two Siblings Affected by Unverricht-Lundborg Disease Using Patient-Derived hiPSCs;Cells;2022-11-04

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