Prolonged high‐glucose exposure decreased SREBP‐1/FASN/ACC in Schwann cells of diabetic mice via blocking PI3K/Akt pathway-Reference-Cited by-同舟云学术

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Prolonged high‐glucose exposure decreased SREBP‐1/FASN/ACC in Schwann cells of diabetic mice via blocking PI3K/Akt pathway

Published:2018-10-26 Issue:4 Volume:120 Page:5777-5789
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J Cell Biochem

Author:

Zhu Lin¹²,Du Wei¹,Liu Yaping¹,Cheng Meijuan¹,Wang Xiumin¹,Zhang Cuihong¹³,Lv Xin¹,Li Fan¹,Zhao Song¹,Hao Jun¹^ORCID

Affiliation:

1. Department of Pathology Hebei Medical University Shijiazhuang Hebei China

2. Department of Electromyogram 3rd Hospital of Hebei Medical University Shijiazhuang Hebei China

3. Department of Radiation Oncology Bethune International Peace Hospital Shijiazhuang Hebei China

Funder

National Natural Science Foundation of China

Natural Science Foundation of Hebei Province

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcb.27864

Reference17 articles.

1. SREBP-1c expression in Schwann cells is affected by diabetes and nutritional status

2. Akt Regulates Axon Wrapping and Myelin Sheath Thickness in the PNS

3. Peripheral nervous system plasmalogens regulate Schwann cell differentiation and myelination

4. Netrin-1 induces the migration of Schwann cells via p38 MAPK and PI3K-Akt signaling pathway mediated by the UNC5B receptor

5. Carboxymethylated chitosan stimulates proliferation of Schwann cells in vitro via the activation of the ERK and Akt signaling pathways

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