Metabolic Investigation on the Interaction Mechanism between Dietary Dihydrochalcone Intake and Lipid Peroxidation Product Acrolein Reduction
Author:
Affiliation:
1. Laboratory for Functional Foods and Human Health Center for Excellence in Post‐Harvest Technologies North Carolina Agricultural and Technical State University North Carolina Research Campus, 500 Laureate Way Kannapolis NC 28081 USA
Funder
National Institute of Food and Agriculture
Publisher
Wiley
Subject
Food Science,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/mnfr.202101107
Reference47 articles.
1. Reactive Carbonyl Species: Diabetic Complication in the Heart and Lungs
2. Reactive Carbonyl SpeciesIn Vivo: Generation and Dual Biological Effects
3. Acrolein and Human Disease: Untangling the Knotty Exposure Scenarios Accompanying Several Diverse Disorders
4. Molecular Mechanisms of Acrolein Toxicity: Relevance to Human Disease
5. Protein-bound acrolein: Potential markers for oxidative stress
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1. Synephrine, as a scavenger and promoter, cooperates with hesperidin to reduce acrolein levels;Food Chemistry;2024-01
2. Detoxification of the Lipid Peroxidation Aldehyde, 4-Hydroxynonenal, by Apple Phloretin In Vitro and in Mice;Journal of Agricultural and Food Chemistry;2023-07-07
3. Synergistic Inhibitory Effect of Multiple Polyphenols from Spice on Acrolein during High-Temperature Processing;Foods;2023-06-09
4. Acrolein: formation, health hazards and its controlling by dietary polyphenols;Critical Reviews in Food Science and Nutrition;2023-05-19
5. Effects of hesperidin combined with synephrine on the capture of acrolein in a mouse model, or in humans by citrus consumption;Food & Function;2023
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