Stable formation of mutated p53 multimers in a Chinese hamster cell line causes defective p53 nuclear localization and abrogates its residual function-Reference-Cited by-同舟云学术

Stable formation of mutated p53 multimers in a Chinese hamster cell line causes defective p53 nuclear localization and abrogates its residual function

Published:2006 Issue:6 Volume:98 Page:1689-1700
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J. Cell. Biochem.

Author:

Ottaggio Laura,Campomenosi Paola,Fronza Gilberto,Menichini Paola,Miele Mariangela,Moro Francesca,Viaggi Silvia,Zunino Annalisa,Abbondandolo Angelo

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference38 articles.

1. Suppression of Human Colorectal Carcinoma Cell Growth by Wild-Type p53

2. Cytoplasmic Accumulation of p53 Protein: an Independent Prognostic Indicator in Colorectal Adenocarcinomas

3. The p53 Status of Chinese Hamster V79 Cells Frequently Used for Studies on DNA Damage and DNA Repair

4. Targeting protein inactivation through an oligomerization chain reaction

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Amplifying Curcumin’s Antitumor Potential: A Heat-Driven Approach for Colorectal Cancer Treatment;OncoTargets and Therapy;2024-01

2. Mechanisms and biological importance of photon-induced bystander responses: do they have an impact on low-dose radiation responses;Journal of Radiation Research;2014-10-31

3. Chemoprevention of rat hepatocarcinogenesis with histone deacetylase inhibitors: Efficacy of tributyrin, a butyric acid prodrug;International Journal of Cancer;2008-12-15

4. A Dominant Role for p53-Dependent Cellular Senescence in Radiosensitization of Human Prostate Cancer Cells;Cell Cycle;2007-03